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The anti-atherosclerosis and attenuated plaque formation effects of GLP-1 RAs are mediated through inhibiting the overall inflammatory response by suppressing the expression and release of proinflammatory cytokines (such as interleukin-6 and tumor necrosis factor-α) and suppressing the activation of nuclear factor-kappa B signaling pathway (23, 24)
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Dose:5mg
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